Mat1-1-3, A Mating Type Gene In Thevillosiclava Virens, Is Required For Fruiting Bodies And Sclerotia Formation, Asexual Development And Pathogenicity
文献类型: 外文期刊
作者: Yong, ML; Yu, JJ; Pan, XY; Yu, MN; Cao, HJ; Qi, ZQ; Du, Y; Zhang, RS; Song, TQ; Yin, XL; Chen, ZY; Liu, WD; Liu, YF
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关键词: Mating Type Gene; Villosiclava Virens; Mat1-1-3; Sexual Development; Pathogenicity
期刊名称: FRONTIERS IN MICROBIOLOGY
ISSN: 1664-302X
年卷期: 2020 年 11 卷
页码:
收录情况: JCR(2021版)
摘要: Villosiclava virensis the prevalent causative pathogen of rice false smut, a destructive rice disease. Mating-type genes play a vital role in the evolution of mating systems in fungi. Some fungi have lostMAT1-1-3, one of the mating-type genes, during evolution, whereas others still retainMAT1-1-3. However, howMAT1-1-3regulates the sexual development of heterothallicV. virensremains unknown. Here, we generated theMAT1-1-3mutants, which exhibited defects in vegetative growth, stress response, pathogenicity, sclerotia formation and fruiting body maturation. An artificial outcrossing inoculation assay showed that the Delta mat1-1-3mutant was unable to produce sclerotia. Unexpectedly, the Delta mat1-1-3mutant could form immature fruiting bodies without mating on potato sucrose agar medium (PSA) compared with the wild-type strain, most likely by activating the truncatedMAT1-2-1transcription to regulate the sexual development. Moreover, RNA-seq data showed that knockout ofMAT1-1-3results in misregulation of a subset of genes involved in sexual development, MAPK signaling, cell wall integrity, autophagy, epigenetic modification, and transcriptional regulation. Collectively, this study reveals thatMAT1-1-3is required for asexual and sexual development, and pathogenicity ofV. virens, thereby provides new insights into the function of mating-type genes in the fungi life cycle and infection process.
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